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The target of hypercapnoea in traumatic brain injury ensures optimal blood flow to the brain.
Thompson’s work comparing ETCO2 and arterial CO2 demonstrated significant disparity between the 2 in a cohort of suspected traumatic brain injury patients?
Kennie’s work looking at a cases load from a single HEMS unit showed equivalent rates of hypoxia and hypotension in those undergoing a PHEA (prehospital emergency anaesthesia)?
Leitch’s work looking at hyperoxia following RSI in a single HEMS unit, showed the majority of patients were hyperoxic according to their ABGs taken within 2 hours of arrival in ED?
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